Which theory of obesity pertains to receptors in the hypothalamus?

The glucostatic theory is focused on the regulation of food intake and energy balance by glucose levels in the bloodstream. This theory posits that the hypothalamus, which is critically involved in regulating hunger and satiety, responds to the concentration of glucose available in the blood. Specifically, when blood glucose levels drop, the hypothalamus triggers hunger signals to encourage food intake. Conversely, when glucose levels are high, it signals satiety, thereby reducing the urge to eat.

Receptors in the hypothalamus play a significant role in monitoring blood glucose levels, as they help to translate changes in glucose availability into physiological responses that drive hunger and energy expenditure. Therefore, the glucostatic theory directly relates to how the hypothalamus functions in response to glucose levels in the body.

While other theories like the lipostatic theory and set point theory also involve the hypothalamus in energy regulation, they focus on different aspects of body weight and energy storage. The lipostatic theory is centered around fat storage and the role of adipocytes in signaling energy reserves, while the set point theory pertains to the body's mechanisms to maintain a certain weight over time, without direct emphasis on glucose levels as a regulator.